https://nova.newcastle.edu.au/vital/access/ /manager/Index en-au 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30059 P for heterogeneity ≥0.36), although associations were strongest in relation to the POAG subtypes with IOP <22 mmHg (MVRR, 1.93; 95% CI, 1.09–3.43) and early paracentral VF loss (MVRR, 2.27; 95% CI, 1.32–3.88). Conclusions: Although the number of natural teeth was not associated with risk of POAG, recent tooth loss was associated with an increased risk of POAG. Because these findings may be due to chance, they need confirmation in larger studies.]]> Tue 03 Oct 2017 13:26:04 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:1896 Sat 24 Mar 2018 08:33:07 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:18053 Sat 24 Mar 2018 08:06:16 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21315 P = 0.0009) but nonsignificant interactions between genetic risk status and weekly fish consumption (P = 0.05) for risk of any AMD. Among participants with high genetic risk, the highest intake tertile of LZ was associated with a >20% reduced risk of early AMD, and weekly consumption of fish was associated with a 40% reduced risk of late AMD. No similar association was evident among participants with low genetic risk. No interaction was detected between ß-carotene or vitamin C and genetic risk status. Conclusions Protection against AMD from greater LZ and fish consumption in persons with high genetic risk based on 2 major AMD genes raises the possibility of personalized preventive interventions.]]> Sat 24 Mar 2018 07:52:47 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21191 Sat 24 Mar 2018 07:52:34 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:5552 Sat 24 Mar 2018 07:49:12 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29629 Sat 24 Mar 2018 07:41:52 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:28445 −2 at rs685352 to 6.4x10⁻⁴ at rs560764, whereas association could not be confirmed for SNPs at the 15q25 locus, with P values ranging from 8.0x10⁻¹ to 6.4x10⁻¹. Ocular biometric analysis revealed that axial length was the most likely trait underlying the refractive error association at the 15q14 locus for SNPs rs560766 (P =0.0054), rs634990 (P =0.0086), and rs8032019 (P =0.0081). Conclusions: Our results confirm the association with refractive error at the 15q14 locus but do not support the association observed at the 15q25 locus. Axial length seemed to be a major parameter at the 15q14 locus, underscoring the importance of this locus in myopia and future clinical treatment. Financial Disclosure(s): The author(s) have no proprietary or commercial interest in any materials discussed in this article.]]> Sat 24 Mar 2018 07:29:01 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:28220 Sat 24 Mar 2018 07:28:30 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:4724 Sat 24 Mar 2018 07:21:50 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:18788 Fri 15 May 2020 06:43:18 AEST ]]>